English class Essay Example | Topics and Well Written Essays - 1000 words

English class - Essay Example Schools can play an important role in educating children about violent crimes prevention. The violent crimes prevention programs gave to be offered to all level of schools. Some programs can be designed in such a way to influence particular age group children, while some can be included in the texts books to archive certain set of goals. Before the implementation of the violent crimes prevention programs, it should be important to justify the quality of the execution, how the programs are executed, what methods are involved in the execution of the crime prevention programs, what effects are expected from the programs, for how long the programs will remain valid, are the programs are focused to achieve short lived goals and how the programs impact the normal lives of the students. The programs are categorized according to the age groups of the student. The general programs are designed for the students with ages from 6 to 17. 3- I think that the increase juvenile crime rate cannot be associated with the schools only. The environment at home also influences the children to indulge in such activities that may harm the personality and behavior of the child. Schools do have a role in training and education children but the initial years the child spends at home may influence him to be violent or behave harshly. Schools are important in determining the needs of the students and impose the violent crime prevention methods according to the behavior of the majority of the students. However, educating the students at very early stage about behaviors will be a better method for the prevention of violent crimes. 4- The source very well explants the research conducted at various stages. The implementation of the programs will lessen the violent juvenile crime rates; however, environment may influence a juvenile to commit a violent crime but it is not such

Effective Teaching Strategies for Students with ADHD Essay Example for Free

Effective Teaching Strategies for Students with ADHD Essay 1. What is the purpose of the study? To share methods of intervention for students with ADHD. 2. When was the study done? 2011 3. Who did the study? George DuPaul 4. What are the research question and hypotheses (if any)? Which school-based intervention strategies are the most effective? Hypothesis: Multiple treatment strategies implemented consistently across multiple years will improve success for students with ADHD. 5. Does the literature background support the need for the study? Please explain your answer. The background literature defines ADHD, explains that these students have issues with retention, attendance and standardized tests. This information provides justification for exploring the topic of effective interventions. 6. What are the variables under study? Identify them as independent, dependent, moderator, control. The independent variables are ADHD and the dependent variable is 7. What is the setting, sample, population? 8. Identify the type of research. Descriptive, non-experimental. 9. What statistical procedures are used, if any? None were used. 10. What kind of design was used? Is it appropriate to the stated purpose, research questions and hypotheses? Since this is a descriptive article, it does not apply any research design nor does it explain the basis of selecting the particular â€Å"effective† strategies. The provision of several strategies does, however provide a variety of effective strategies to implement, which supports the opening statement, that it takes consistent implementation of multiple effective strategies for successful intervention with ADHD students. 11. What are the findings of the study? As stated above, the study impresses that it takes consistent implementation of multiple effective strategies for successful intervention with ADHD students. 12. Are there any problems with the internal validity of this study? The fact that the students are from a socioeconomically disadvantaged background and that the control and experimental groups were selected at random increases my confidence that there is internal validity. 13. Are there any problems with the external validity of this study? It is not possible to say that this study necessarily applies to the population because it was only conducted in one classroom. If there was a more robust sample from a variety of schools I would feel more confident applying this to the whole population in general. 14. What can be validly concluded from this study? The fact that there is no explanation for the choices that were made for â€Å"effective† strategies leads me to say that this paper is merely descriptive of options, not necessarily of the most effective interventions for ADHD.

Social Responsibilities :: essays research papers fc

Social Responsibilities: 1.  Ã‚  Ã‚  Ã‚  Ã‚  Health Aspect a.  Ã‚  Ã‚  Ã‚  Ã‚  Find better ways to cut down on fat and calories Is Starbucks adding to the growing weight problem with American society? You be the judge. I personally was shocked at the nutritional facts on their products (Figure 1). Some fast food places offer healthier products than some of the Starbucks products (Figure 2). The nutritional facts circled in red in figure 1 and figure 2 shows that one cup of Starbucks Pumpkin Spice Crà ¨me with whip is less nutritional than an entire meal at McDonalds. Imagine what the nutritional facts would look like if you included a Classic Coffee Cake with your coffee beverage! (Figure 1.2)   Ã‚  Ã‚  Ã‚  Ã‚  Ã‚  Ã‚  Ã‚  Ã‚  Ã‚   The Starbucks nutritional values listed below are not completely accurate as shown in the following statement: â€Å"The nutrition information for beverages is based on Starbucks ® standardized beverage recipes. Because each of our beverages is handcrafted and may be customized, some variation can be expected. The data is calculated using ESHA Research’s Genesis ® R&D software. All data is rounded to meet current United States Food and Drug Administration (FDA) packaged foods labeling regulations.† Works cited: (http://www.starbucks.com/retail/nutrition_info.asp) This means that depending on your taste, the fat content and calories could be more than what is shown in the chart. Figure 1 – Starbucks Nutritional Facts (Beverages) (http://www.starbucks.com/retail/nutrition_info.asp) Figure 1.2 – Starbucks Nutritional Facts (Food Items) (http://www.starbucks.com/retail/nutrition_freshfood_detail.asp) Figure 2 – McDonalds Nutritional Facts (http://app.mcdonalds.com/bagamcmeal) What is Starbucks planning to do to lower fat and calories in their products? Starbucks is offering non-fat milk and soy to help trim down the fat and calories. The alternative ingredients do alter the flavor a bit, so it may not be the choice of most customers. b.  Ã‚  Ã‚  Ã‚  Ã‚  Advertise to public about fat content of their products It seems like Starbucks doesn’t want the general public to really know how bad the nutritional facts are right up front in their stores. If you walk into a Starbucks store you see mostly pictures of some of their least nutritional coffee beverages because they look the most appetizing. I think if more people knew how bad Starbucks products were they would see a significant reduction in revenue. This would be especially true with today’s low carb, low fat health trends. Starbucks does however provide a nice nutritional facts chart on their website, but who is looking at the website while ordering anyway? So as far as I can tell Starbucks plans on feeding America’s sweet tooth and will continue to fatten them up.

The Role of Inflammation in the Advancement of Chronic Obstructive

The Role of inflammation in the advancement of Chronic Obstructive Pulmonary disease. Introduction Chronic obstructive pulmonary disease (COPD) is the collective term used for respiratory disease, including chronic bronchitis and emphysema. The disease develops slowly and is often not diagnosed until it is advanced and irreparable damage is evident (Global Initiative for Chronic Obstructive Lung Disease, 2011). The disease is characterised by airflow obstruction and lung parenchyma.Parenchyma, associated with emphysema, is the permanent enlargement of the air spaces distal to the terminal bronchioles, accompanied by airway wall destruction, without obvious fibrosis (Demirjian and Kamangar, 2011; Atsuyasu et al. , 2007). Airflow limitation results from loss of elastic recoil and reduced airway tethering. Chronic bronchitis leads to narrowing of airway calibre, increasing airway resistance. Patients may display signs of one or both of these diseases as they frequently occur in associat ion with each other.Common symptoms are wheezing, coughing, shortness of breath on exertion, production of sputum and recurrent respiratory infections (Global Initiative for Chronic Obstructive Lung Disease, 2011). There are a host of triggers that exacerbates symptoms including smoking and environmental pollutants, resulting in chronic inflammation (Kazuhiro and Barnes, 2009; Manuel et al. , 2002). â€Å"Inflammation is defined as the presence of redness, swelling and pain, caused by the presence of edema fluid and the infiltration of tissues by leukocytes† (Nairn & Helbert, 2002, pp15).Inflammation is a key biological response to eliminate harmful pathogens, but there is increasing evidence to suggest that chronic inflammatory responses are accountable for the advancement of this disease and other chronic diseases including coronary artery disease, cancer, rheumatoid arthritis and multiple sclerosis. This review explores the correlation between COPD and inflammation and the subsequent effects on the systemic systems and the link with coronary heart disease (Mantovini et. al. , 2008; Mohr & Pelletier, 2005; Sattar et. al. , 2003; Powells et. al. , 2001; Danesh et. al. 2000; Murdoch & Finn, 2000). Methods Search engines used were Google Scholar and Pub Med using the keywords COPD, inflammation, disease, apoptosis, interleukin 8, cytokines, coronary heart disease and COPD. Searches were restricted to dates between 1999 and 2012. The majority of the included papers were obtained from the reference lists of other research papers. COPD risk factors: COPD is strongly linked with repeated exposure to noxious particles or gases and cigarette smoke has been acknowledged as a prime risk factor (Fabri et. al. , 2006; Lindberg et al. , 2005; Pauwels and Rabe. 2004, Association for Respiratory Technology & Physiology, 2000). Smokers have an increased prevalence of respiratory and lung function abnormalities, a greater rate of decline in FEV1 and a higher mortality rate than non-smokers (World health organisation, 2012). However, only a third of smokers develop COPD which implies that other factors such as genetics and environment are involved (Agusti, 2003). Exposure to air pollution caused by heating and cooking with bio-mass fuels in poorly ventilated housing are major risk factors for COPD, especially in developing countries (Pauwels & Rabe, 2004).The most documented COPD genetic risk factor is the deficiency of Alpha -1-antitrypsin, a polymorphic glycoprotein which offers anti-protease protection against the serine proteinease, neutrophil elastase (Abboud & Vimalanathan, 2008; Devereux, 2006; Siafakas & Tzortzaki, 2002; Fabbri et al. , 2006). Research studies (in vitro) indicated that Alpha – 1 – antitrypsin also possesses anti-inflammatory capabilities that extend beyond its anti-protease role, including regulation of CD14 expression (Nita, Serapinas & Janciauskiene, 2007), inhibition of TNF-? ene upregulation (Subramaniyam , 2007) and inhibition of lipopolysaccharide activation of monocytes and neutrophil migration (Janciauskiene et al. , 2004). Deficiency of Alpha -1-antitrypsin is associated with COPD progression in both smokers and non-smokers, although far greater in smokers (Bergen et al. , 2010; Fabbri et al. , 2006; Siafakas and Tzortzaki. , 2002; Foos et al. , 2002). Studies have suggested that smoking with this genetic disposition will substantially increase risk of developing COPD (Kohnlein & Welte, 2008; Pauwels & Rabe, 2004; Foos et al. , 2002; Siafakas & Tzortzaki, 2002; Association forRespiratory Technology and Physiology, 2000). Pathogenesis of COPD Exposure to noxious particles â€Å"†¦ triggers cytokine activation to recruit cells, which play a vital role in removing the noxious agents†¦ † (Nairn & Helbert, 2007, pp22). An infiltration of neutrophils, eosinophils and CD8+ T-lymphocytes into the airways and lungs follows (Demedts et al, 2006; Mahler et al. , 2004; Sopo ri, 2002). High concentrations of chemokines, interleukon-8 (IL8) and tumor necrosis factor-a have been found in patients with COPD which are potent activators and chemo-attractants of leukocyte subpopulations (Murdoch and Finn, 2000; Yamamoto et al. 1997). The interaction of chemo-attractants with leukocytes initiates a series of coordinated and cellular events, which includes phagocytosis, release of soluble anti-microbials and formation of reactive oxygen compounds involved in intracellular killing (Murdoch & Finn, 2000). Neutrophils and macrophages release elastase, stimulating the production of mucus to assist in ridding the airways of the irritants and subsequent waste generated by the inflammatory response (Shimizu et al. , 2000).Other processes such as neutrophil necrosis and reactive oxygen species further contribute to mucus hyper secretion (Kim and Nadel, 2004; Mizgerd, 2002). When an inflammatory response is no longer needed protease inhibitor cells dampen the response. Research suggests that the inhibiting response in COPD is not triggered and chronic inflammation presides, representing a crucial mechanism in the pathogenesis of COPD (Demedts et al. , 2006; Hodge et al 2004). Hypersecretion of mucous can inhibit the ciliated epithelium from transporting mucus from the airways.Subsequent delays in bacteria clearance results in bacterial colonisation, which stimulates further granulocytic recruitment to the airways, escalating the inflammatory response. Chronic inflammation is linked with tissue destruction, imbalance of proteolytic and anti-proteolytic activity, hyper secretion of mucus, increased apoptotic activity and oxidative stress which contribute to the progression of COPD. Long term, chronic inflammation can result in widespread airway and parenchymal cell destruction which further contributes to disease progression (Mantovini et al. 2008; Mohr and Pelletier, 2005; Sattar et al. , 2003; Sopori, 2002; Powells et al. , 2001; Danesh et al. , 2 000; Murdoch & Finn, 2000). Research suggests that macrophages express a markedly lower amount of toll like receptors in COPD suffers, resulting in a decreased recognition of microbes, facilitating damaging microbial colonisation, which may explain the increased amount of respiratory infections in COPD sufferers (Schneberger, 2011; Droemann et al. 2005). Infection initiates a biased release of inflammatory mediators which may escalate the pathogenesis of the disease (Gaschler et al. 2009, Ritter et al. , 2005; Sethi, 2000). Oxidative stress Demedts et al, 2005 found that the alveolar macrophages of COPD sufferers produced much higher levers of oxygen radicals and myeloperoxide which are important for the destruction of inter-cellular pathogens. Oxidant/anti-oxidant imbalance can result in the inactivation of anti-proteinases, airspace epithelial injury, increased sequestration of neutrophils in the pulmonary microvasculature, and gene expression of pro-inflammatory mediators, all of which exacerbate the inflammatory response (MacNee, 2000 Drost et al. 2005). Emphysema like changes have been show in the CT scans of malnourished women, suggesting that diet has an effect on lung tissue in the absence of smoking (Coxon et al. , 2004). Dietary supplementation then may be a beneficial therapeutic intervention in this condition, as antioxidants not only protect against the direct injurious effects of oxidants, but fundamentally alter the inflammatory events that play an important part in the pathogenesis of COPD (Coxon et al. , 2004; MacNee, 2000). Apoptosis and COPDResearch suggests that there is increased apoptosis of epithelial cells in smokers and COPD patients. Apoptosis persisted despite smoking cessation which suggests apoptosis may play a part in driving the inflammatory process and progression of the disease (Hodge et al. , 2003). Increased apoptotic alveolar epithelial and endothelial cells in the lungs not counterbalanced by proliferation and sufficient ph agocytic clearance results in destruction of lung tissue and development of emphysema (Demedts et al, 2006; Kazutetsu, Naoko & Atsushi, 2003; Barnes et al. 000) Apoptosis can be induced by various stimuli, including oxidative stress, elastase and infiltrating cytoxix CD8 + T cells which are all associated with inflammation (Kazutetsu, Naoko and Atsushi, 2003). Efferocytosis allows for the removal of apoptotic material with minimal inflammation and prevents the development of secondary necrosis and ongoing inflammation. Failure of this highly conserved process may contribute to disease pathogenesis by impeding both the resolution of inflammation and the maintenance of alveolar integrity (Mukaro and Hodge, 2011; Taylor et al. , 2010; Morimoto et al, 2006; Vandivier et al, 2006).Proteolytic/Anti-proteolytic activity Mukaro and Hodge, (2011) suggests that in COPD there is an imbalance between proteolytic and anti-proteolytic activity, a prominent factor in the pathogenesis of this disea se, which may contribute to lung parenchymal destruction. Research has also found that macrophages demonstrate defective phagocytic ability against common airways pathogens in COPD (Taylor et al. , 2010; Hodge et al. , 2003), The findings of Berenson et al. , (2006), supported a paradigm of defective immune responsiveness of alveolar macrophages, but found no significant differences in the blood macrophages of COPD sufferers.Taylor (2010) believes that persistence of bacteria as a consequence of defective phagocytosis may be a chronic antigenic drive for chronic inflammation. Systemic effects of COPD â€Å"Chronic inflammation is present in all disease processes, mediating all stages of disease from initiation, manifestation and maturation† (Sompayrac 2003, pp12). Compelling epidemioligical data links systemic inflammation to atherosclerosis, ischemic heart disease, strokes, and coronary deaths (Danesh, Whincup and Walker, 2000; Ridker, 1999).These observations have been stro ngly supported by experiments that show the direct effects of certain inflammatory markers, such as C-reactive protein (CRP), on the pathogenesis of plaque formation (Zwaka, Hombach and Torzewski, 2001; Lagrand, Visser & Hermens, 1999). A study by Gan, Man & Sin, 2003) found that patients with COPD were 2. 18 times more likely to have an elevated circulating c-reactive protein levels. Evidence strongly suggests that there is relationship between COPD, systemic inflammation, and cardiovascular diseases.Studies show that patients with mild-to-moderate COPD, cardiovascular disease is the leading cause of morbidity and mortality (Din and Man, 2009; Pope et al, 2003). As these diseases share similar risk factors such as smoking, increased age and inactivity, causation is unclear and is likely to be due to multiple factors, including lifestyle, environmental and genetics (Gan, 2005; Agusti et. al. 2003). Discussion Inflammation, it would appear, is a double edged sword; crucial for cleara nce of pathogens and recovery from injury; but can also contribute to life threatening chronic diseases (Smith, 1994; Sporori, 2003).COPD is a complex condition, influenced by multiple genetic and/or environmental risks. A cycle of low grade inflammation is the consequence, with destructive and damaging effects, resulting in mucus hyper-secretion, airway obstruction, increased elastase production and oxidative stress, which encourage further inflammation and destruction. COPD is associated with exposure to smoke or noxious gases, however inflammation may also be caused by irritation from coughing, wheezing, respiratory infections and mucus production. Most exacerbations of COPD are caused by bacterial or viral infection (Sanjay and Murphy, 2008; Sanjay 2008).Mucosal cells produce mucus, which irritates the airways causing airway obstruction. This subsequently reduces FEV1, and cough effectiveness, which contributes to the build up of bacterial mucus. Imbalance between proteolytic an d anti-proteolytic activity presides, creating an ideal environment for infection. Research suggests that macrophages express a markedly lower amount of toll like receptors in COPD suffers, resulting in a decreased recognition of microbes, facilitating damaging microbial colonisation, which may explain the increased amount of respiratory infections in COPD sufferers (Schneberger,2011; Droemann et al. 005). Infection initiates a biased release of inflammatory mediators which may escalate the pathogenesis of the disease (Gaschler et al. , 2009, Ritter et al. , 2005; Sethi, 2000). Researchers have found high levels of neutrophils, macrophages and CD8+ cells in ex smokers (Lappers et al. , 2006). Thus, suggesting that inflammatory changes in COPD, although initially induced by inhalation of noxious agents, are fundamental to the disease process, rather than to the initial trigger per se (Gamble et al, 2007). Studies have shown that airway epithelial and T-cell apoptosis in COPD continue s despite smoking cessation (Lappers et al. 2006). Excess apoptosis results in inappropriate destruction of host tissue, leading to atrophy and tissue necrosis, which in turn further stimulates the inflammatory response and perpetuates the situation. We have already ascertained an imbalance between the proteolytic and anti-proteolytic activity and this is another factor that contributes, resulting in failure to resolve the inflammatory reaction rapidly (Hodge et al. , 2005). Un-cleared apoptotic cells may undergo secondary necrosis with discharge of injurious cells contents resulting in tissue destruction and further inflammation.Inability to remove apoptotic cells and debris created overwhelms the normal clearance mechanisms, stimulating further inflammatory responses, further contributing to COPD pathogenesis (Sanjay and Murphy, 2008; Sanjay 2008). It has been identified that the immune system may become less responsive, the longer that chronic inflammation presides, which may lea d you to believe that this would initiate an inhibitory effect on the inflammatory process. However this is not the case and the inflammatory process persists, presenting as low level chronic inflammation.In addition a less responsive immune system is more susceptible to infection, exacerbating the inflammatory response (Sanjay and Murphy, 2008; Sanjay 2008). There appears to be strong epidemiological links between cardiovascular disease and COPD. The same inflammatory markets are evident in both suggesting a systemic link. Both diseases share similar risk factors, so it is difficult to determine initiation of the diseases. One could also argue that the debilitating effects of COPD, which include a reduced exercise capacity, dyspnoea and deconditioning increase the risk of cardiovascular disease development.In conclusion, it appears that adaptive immune is active in the disease progression of this complex pathophysiological syndrome. Particularly elaboration and production of cytoki nes, chemical mediators and auto-antibodies, which directly injure respiratory tissues. CD8+ mediates tissue destruction, whereas CD4 orchestrates inflammatory responses, which facilitates humoral immune responses (Gadgill and Duncan 2008). Conclusions made in this review are only valid within the boundaries of the research and papers used. 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Southwest Airlines: Organizational Commitment and Communication

Southwest Airlines’ strong organizational culture is reflected in its mission as shown in its website: â€Å"dedication to the highest quality of Customer Service delivered with a sense of warmth, friendliness, individual pride, and Company Spirit. † Southwest is a company that not only excels in customer service and profitability but also in employee development. Its organizational commitment is not reduce to the organization; it is actually expand it to an ongoing relationship with the employees where they have the opportunity to express their ideas, suggestions and concerns. This organizational culture has made Southwest Airlines one of the companies with the fewest customer complaints, and an employee turnover ratio of less than 10 percent (D’Aurizio, 2008). The same strong culture Southwest Airlines has built over the time is supported by every employee who has in mind that they have to deliver a warm and friendly assistance to their customers. Southwest management’s strategy could not be any better; the management treats employees as family, expecting from the employees to treat customers like family as well. The company is characterized for having a decentralized structure. In between the CEO and the frontline supervisors, there are only a few levels. Their philosophy on structure is very relaxed. Any employee who feels the need is welcomed to contact someone above their supervisor. The president has three executive vice presidents who each have five or six vice presidents who report to them. There is one vice president of Internal Audits and Special Projects who reports directly to the president. (Rivera, Cornwell, Abenes, 2003) The Southwest culture creates and integrates a dedicated and motivated workforce that succeeds together. The owners’ leadership style is led by example; that is how Kelleher treats his employees, like family, he is open to communicate and he also knows the vital role in forming this unique culture. Before people become employed at Southwest, the managers in charge of the hiring interview make sure they embed the organizational culture in the candidates. When they become part of the Southwest family, then they are treated like part of it. Any event in the lives of the employees are acknowledged and celebrated by everyone, everyone cherish for the success and happiness of the members of the family. The company establishes special programs for families of employees. The company listens to comments and feedback, not only from employees, but also employees’ families, customers, and vendors (D’Aurizio, 2008). This company has become profitable by putting into practice the philosophy of an open communication. Southwest does not have the culture only in paper, they actually live it. The managers start giving the example to the employees, and they follow their leaders. Southwest strategically generates satisfaction to their employees, this needs satisfactions will later reflect and convert into satisfaction to the customer and shareholder value via organizational capabilities I believe the success of Southwest Airlines comes from the combination of different sources of power. Part of it comes from the expert power, and also from the reward power. Southwest management had has the ability and knowledge to create such an amazing organizational culture, this ability is the expertise power. Now the way they have applied the idea into real actions, the fact that they led by example and treat the employees as family is the reward power. The management has gained the respect, trust and loyalty from their employees. This is because of what they have done not only with words but more important with actions. Concerning the communication aspect, this plays an important role in the organizational culture of Southwest Airlines and the way the employees perceive that culture. The website Reference for Business (2009) explain how the contend of a message send will be perceive in different way by every individual. Each person gives their own personal meaning to the message received. This also happens due to the experience, the receiver makes sense out of a message based on experience. No one can perceive experiences in the same exact way as another does. Each individual has a unique set of experiences, a unique perceptual â€Å"filter,† that helps them to understand or interpret messages received. These filters are built over time by each individual person according to their childhood background, and the life experiences. Finally, we can say that the combination of the message received and he experience of the receiver is what makes an interpretation of the message. This is the reason why Southwest management emphasizes in a clear and concrete communication. They know how some messages can be easily misunderstood or twisted on the way. And this is the reason why they keep an open communication to the employees. Allowing questions when the message was not clear, as well as allowing suggestions when the employees do not agree. The challenge for every organization is how to create a great channel of communication that will enable employees to comprehend messages in the way they were originally intended to be transmitted. According to Beebe and Masterson, â€Å"conflict in a group occurs when members disagree over two or more options that a group can take in trying to make a decision, resolve a problem, or achieve a goal† (Chapter 7, p. 69). Other types of conflicts can occur as a result of differences in goal. Now, the good news about conflict is that it can be use in a positive way contrarily to the popular belief that states that a conflict should always be avoided. Southwest Airlines, for example, could use conflict to challenge their employees to research, or come up with ideas of how to resolve some specific issues, problems. Additional researches will help to determine a better way to fix problem that are costing a lot of money to the company. The commitment of Southwest airlines is to a culture that motivates, empowers and promotes employees to make the company one of the best companies to work for, as well as one of the most profitable one. One of the most important tools this company uses is the open communication and the in-house advancement that encourages a positive perception of company culture, allowing employees the opportunity to express ideas, suggestions, concerns that Make them believe they are part of the Southwest â€Å"family†. Southwest best strategy is the commitment not only to the organization itself but also to the employees. Everyone there is part of a big family who works hard to keep harmony n within the family. They use great leadership styles where the managers led by example and they are always open to any communication. This company should be use as example as what it is to build a great culture and a great organizational commitment.